Decursin inhibits the growth of HeLa cervical cancer cells through PI3K/Akt signaling-Reference-Cited by-同舟云学术

Decursin inhibits the growth of HeLa cervical cancer cells through PI3K/Akt signaling

Published:2020-11-25 Issue:6 Volume:23 Page:584-595
ISSN:1028-6020
Container-title:Journal of Asian Natural Products Research
language:en
Short-container-title:Journal of Asian Natural Products Research

Author:

Zhu Meng-Lian¹²³,Li Jian-Chun³,Wang Lu³⁴,Zhong Xia³,Zhang Yu-Wei³,Tan Rui-Zhi³,Wang Hong-Lian³,Fan Jun-Ming²³⁵,Wang Li³

Affiliation:

1. Nosocomial Infection Control Department, Chongqing Jiangbei Hospital of Traditional Chinese Medicine, Chongqing 400000, China

2. Department of Nephrology, The Affiliated Traditional Medicine Hospital of Southwest Medical University, Luzhou 646000, China

3. Laboratory of Organ Fibrosis Prophylaxis and Treatment, Research Center of Integrated Chinese and Western Medicine, Affiliated Traditional Medicine Hospital of Southwest Medical University, Luzhou 646000, China

4. Department of Nephrology, The Affiliated Hospital of Southwest Medical University, Luzhou 646000, China

5. Chengdu Medical College, Chengdu 610500, China

Funder

Sichuan provincial Health Commission

The Basic Research Programs of Science and Technology Commission Foundation of Sichuan Province

Technology and Human resources’ Bureau of Luzhou

The Youth Funding Program of Southwest Medical University

Publisher

Informa UK Limited

Subject

Organic Chemistry,Complementary and alternative medicine,Drug Discovery,Pharmaceutical Science,Pharmacology,Molecular Medicine,General Medicine,Analytical Chemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/10286020.2020.1821669

Reference17 articles.

1. Safety against nephrotoxicity in paclitaxel treatment: Oral nanocarrier as an effective tool in preclinical evaluation with marked in vivo antitumor activity

2. Dose and schedule-finding study of oral topotecan and weekly cisplatin in patients with recurrent ovarian cancer

3. Decursin and decursinol angelate inhibit estrogen-stimulated and estrogen-independent growth and survival of breast cancer cells

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