Growth control of multiple myeloma cells through inhibition of glycogen synthase kinase-3
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
http://www.tandfonline.com/doi/pdf/10.1080/10428190802304966
Reference44 articles.
1. Understanding multiple myeloma pathogenesis in the bone marrow to identify new therapeutic targets
2. Interleukin-6–Induced Inhibition of Multiple Myeloma Cell Apoptosis: Support for the Hypothesis That Protection Is Mediated Via Inhibition of the JNK/SAPK Pathway
3. IL-6 up-regulates Mcl-1 in human myeloma cells through JAK / STAT rather than Ras / MAP kinase pathway
4. Insulin-like growth factor I is a dual effector of multiple myeloma cell growth
5. IGF-1 receptor tyrosine kinase inhibition by the cyclolignan PPP induces G2/M-phase accumulation and apoptosis in multiple myeloma cells
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