Activation of Protein Kinase C Enhances TNF-α-induced Differentiation by Preventing Apoptosis via Rapid up-regulation of c-Myc Protein Expression in HL-60 Cells
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
http://www.tandfonline.com/doi/pdf/10.1080/1042819021000047010
Reference38 articles.
1. Differential biologic effects resulting from bimodal binding of recombinant human tumor necrosis factor to myeloid leukemia cells
2. The type B receptor for tumor necrosis factor-alpha mediates DNA fragmentation in HL-60 and U937 cells and differentiation in HL-60 cells
3. c-Myc and Bcl-2 Protein Expression During the Induction of Apoptosis and Differentiation in TNFα-Treated HL-60 Cells
4. TRADD–TRAF2 and TRADD–FADD Interactions Define Two Distinct TNF Receptor 1 Signal Transduction Pathways
5. TNF Receptor Death Domain-associated Proteins TRADD and FADD Signal Activation of Acid Sphingomyelinase
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1. Production of porcine TNFα by ADAM17-mediated cleavage negatively regulates porcine reproductive and respiratory syndrome virus infection;Immunologic Research;2016-01-02
2. Tumor necrosis factor-α enhances DMSO-induced differentiation of HL-60 cells through the activation of ERK/MAPK pathway;International Journal of Hematology;2008-02-08
3. Neutrophil and monocyte receptor expression in uncomplicated and complicated influenza A infection with pneumonia;Scandinavian Journal of Infectious Diseases;2008-01
4. The Protein Kinase C (PKC) Family of Proteins in Cytokine Signaling in Hematopoiesis;Journal of Interferon & Cytokine Research;2007-08
5. Granulocyte differentiation inducer, hexapeptide HLDF-6, decreases cytotoxic effect of tumor necrosis factor on HL-60 cell line;Biochemistry (Moscow);2007-01
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