CCR5 expression in inflammatory bowel disease and its correlation with inflammatory cells and β-arrestin2 expression-Reference-Cited by-同舟云学术

CCR5 expression in inflammatory bowel disease and its correlation with inflammatory cells and β-arrestin2 expression

Published:2017-01-31 Issue:5 Volume:52 Page:551-557
ISSN:0036-5521
Container-title:Scandinavian Journal of Gastroenterology
language:en
Short-container-title:Scandinavian Journal of Gastroenterology

Author:

Ye Xiaoyan¹²,Liu Sixue¹³,Hu Mei¹³,Song Yangda¹³,Huang Huarong³⁴,Zhong Yingqiang¹³

Affiliation:

1. Department of Gastroenterology and Hepatology, Sun Yat-Sen Memorial Hospital of Sun Yat-Sen University, Guangzhou, China;

2. Department of Gastroenterology and Hepatology, the First Affiliated Hospital of Guangdong Pharmaceutical University, Guangzhou, China;

3. Key Laboratory of Malignant Tumor Gene Regulation and Target Therapy of Guangdong Higher Education Institutes of Sun Yat-Sen University, Guangzhou, China;

4. Department of Pediatrics, Sun Yat-Sen Memorial Hospital of Sun Yat-Sen University, Guangzhou, China

Funder

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Publisher

Informa UK Limited

Subject

Gastroenterology

Link

https://www.tandfonline.com/doi/pdf/10.1080/00365521.2017.1281435

Reference40 articles.

1. The Chemokine RANTES Is a Crucial Mediator of the Progression from Acute to Chronic Colitis in the Rat

2. Treatment with Met-RANTES decreases bacterial translocation in experimental colitis

3. The simultaneous blockade of chemokine receptors CCR2, CCR5 and CXCR3 by a non-peptide chemokine receptor antagonist protects mice from dextran sodium sulfate-mediated colitis

4. Chemokine receptor homo- or heterodimerization activates distinct signaling pathways

5. Crucial involvement of the CCL3-CCR5 axis-mediated fibroblast accumulation in colitis-associated carcinogenesis in mice

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