Secondary enucleated retinoblastoma with MYCN amplification
Author:
Affiliation:
1. Jules-Gonin Eye Hospital, Lausanne University, Lausanne, Switzerland
2. Oncogenomic Laboratory, CHUV, Lausanne, Switzerland
3. Pediatric Oncology, CHUV, Lausanne, Switzerland
Funder
no funding
Publisher
Informa UK Limited
Subject
Genetics (clinical),Ophthalmology,Pediatrics, Perinatology and Child Health
Link
https://www.tandfonline.com/doi/pdf/10.1080/13816810.2021.1897847
Reference24 articles.
1. N-MYC amplification, loss of heterozygosity on the short arm of chromosome 1 and DNA ploidy in retinoblastoma
2. N-myc amplification was rarely detected by fluorescence in situ hybridization in retinoblastoma
3. High level amplification of N-MYC is not associated with adverse histology or outcome in primary retinoblastoma tumours
4. Phosphorylation of pRb: mechanism for RB pathway inactivation inMYCN-amplified retinoblastoma
5. Characterisation of retinoblastomas without RB1 mutations: genomic, gene expression, and clinical studies
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1. Ceftriaxone exerts antitumor effects in MYCN‐driven retinoblastoma and neuroblastoma by targeting DDX3X for translation repression;Molecular Oncology;2023-11-27
2. Effect of CDK7 inhibitor on MYCN-amplified retinoblastoma;Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms;2023-09
3. MRI Features for Identifying MYCN-amplified RB1 Wild-type Retinoblastoma;Radiology;2023-06-01
4. Genetic Counseling;Pediatric Vitreoretinal Surgery;2023
5. An immature, dedifferentiated, and lineage-deconstrained cone precursor origin of N-Myc–initiated retinoblastoma;Proceedings of the National Academy of Sciences;2022-07-08
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