Reduction of NO- and EDHF-Mediated Vasodilatation in Hypertension: Role of Asymmetric Dimethylarginine
Author:
Publisher
Informa UK Limited
Subject
General Medicine,Physiology,Internal Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1080/10641960701616194
Reference33 articles.
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2. Relative Contributions of NO and Gap Junctional Communication to Endothelium-Dependent Relaxations of Rabbit Resistance Arteries Vary with Vessel Size
3. Aged Spontaneously Hypertensive Rats Exhibit a Selective Loss of EDHF-Mediated Relaxation in the Renal Artery
4. Display of the characteristics of endothelium-derived hyperpolarizing factor by a cytochrome P450-derived arachidonic acid metabolite in the coronary microcirculation
5. Characterization of endothelium-derived hyperpolarizing factor as a cytochrome P450-derived arachidonic acid metabolite in mammals.
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1. Vascular adenosine monophosphate‐activated protein kinase: Enhancer, brake or both?;Basic & Clinical Pharmacology & Toxicology;2019-11-24
2. Endothelium-Dependent Hyperpolarization (EDH) in Hypertension: The Role of Endothelial Ion Channels;International Journal of Molecular Sciences;2018-01-21
3. Menthol-Induced Cutaneous Vasodilation Is Preserved in Essential Hypertensive Men and Women;American Journal of Hypertension;2017-07-26
4. Development of an Adverse Outcome Pathway for the Onset of Hypertension by Oxidative Stress-Mediated Perturbation of Endothelial Nitric Oxide Bioavailability;Applied In Vitro Toxicology;2017-03
5. Nebivolol ameliorates asymmetric dimethylarginine-induced vascular response in rat aorta via β3adrenoceptor-mediated mechanism;Clinical and Experimental Hypertension;2016-01-29
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