Interdependency of <i>NINJ2</i> gene expression and polymorphism with susceptibility and response to interferon beta in patients with multiple sclerosis-Reference-Cited by-同舟云学术

Interdependency of NINJ2 gene expression and polymorphism with susceptibility and response to interferon beta in patients with multiple sclerosis

Published:2022-10-20 Issue: Volume: Page:1-6
ISSN:0020-7454
Container-title:International Journal of Neuroscience
language:en
Short-container-title:International Journal of Neuroscience

Author:

Khorrami Mehdi¹^ORCID,Saneipour Maryam²^ORCID,Moridnia Abbas²^ORCID,Shaygannejad Vahid³^ORCID,Sadeghi Erfan⁴^ORCID,Kassani Aziz⁵^ORCID,Sarmadi Akram¹^ORCID,Mirmosayyeb Omid³^ORCID

Affiliation:

1. Department of Genetics and Molecular Biology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran

2. Department of Genetics and Molecular Biology, School of Medicine, Dezful University of Medical Sciences, Dezful, Iran

3. Isfahan Neuroscience Research Center, Isfahan University of Medical Sciences, Isfahan, Iran

4. Department of Biostatistics and Epidemiology, Faculty of Health, Isfahan University of Medical Sciences Isfahan Iran, Isfahan, Iran

5. Department of Community Medicine, School of Medicine, Dezful University of Medical Sciences, Dezful, Iran

Funder

Dezful University of Medical Sciences

Publisher

Informa UK Limited

Subject

General Medicine,General Neuroscience

Link

https://www.tandfonline.com/doi/pdf/10.1080/00207454.2022.2102979

Reference19 articles.

1. Multiple sclerosis

2. Evidence of an increased prevalence of multiple sclerosis: a population-based study of Tehran registry during 1999–2018

3. Inferring Multiple Sclerosis Stages from the Blood Transcriptome via Machine Learning

4. Approved and Emerging Disease Modifying Therapies on Neurodegeneration in Multiple Sclerosis

5. Response to interferon-beta treatment in multiple sclerosis patients: a genome-wide association study

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4. Involvement of NINJ2 Protein in Inflammation and Blood–Brain Barrier Transmigration of Monocytes in Multiple Sclerosis;Genes;2022-10-25