Gilteritinib for the treatment of patients withFLT3mutated relapsed or refractory acute myeloid leukemia
Author:
Affiliation:
1. Leukemia Service, Department of Medicine, Roswell Park Comprehensive Cancer Center, Buffalo, NY, USA
Funder
National Cancer Institute
Roswell Park Alliance Foundation
Publisher
Informa UK Limited
Subject
Drug Discovery,Pharmacology,Genetics,Molecular Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/23808993.2019.1612709
Reference28 articles.
1. The roles of FLT3 in hematopoiesis and leukemia
2. Downstream molecular pathways of FLT3 in the pathogenesis of acute myeloid leukemia: biology and therapeutic implications
3. Genomic instability is a principle pathologic feature of FLT3 ITD kinase activity in acute myeloid leukemia leading to clonal evolution and disease progression
4. Profiling of somatic mutations in acute myeloid leukemia with FLT3-ITD at diagnosis and relapse
5. Phase IIB Trial of Oral Midostaurin (PKC412), the FMS-Like Tyrosine Kinase 3 Receptor (FLT3) and Multi-Targeted Kinase Inhibitor, in Patients With Acute Myeloid Leukemia and High-Risk Myelodysplastic Syndrome With Either Wild-Type or Mutated FLT3
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