A method comparison in monitoring disease progression of G93A mouse model of ALS
Author:
Publisher
Informa UK Limited
Subject
Clinical Neurology,Neurology,General Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1080/17482960701538759
Reference18 articles.
1. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis
2. UNRAVELING THE MECHANISMS INVOLVED IN MOTOR NEURON DEGENERATION IN ALS
3. Motor Neuron Degeneration in Mice that Express a Human Cu,Zn Superoxide Dismutase Mutation
4. Thalidomide and Lenalidomide Extend Survival in a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis
5. Wild-type microglia extend survival in PU.1 knockout mice with familial amyotrophic lateral sclerosis
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2. Assessing the therapeutic impact of resveratrol in ALS SOD1-G93A mice with electrical impedance myography;Frontiers in Neurology;2022-12-22
3. Muscle contractility dysfunction precedes loss of motor unit connectivity in SOD1(G93A) mice;Muscle & Nerve;2018-12-21
4. Expression of a Mutant SEMA3A Protein with Diminished Signalling Capacity Does Not Alter ALS-Related Motor Decline, or Confer Changes in NMJ Plasticity after BotoxA-Induced Paralysis of Male Gastrocnemic Muscle;PLOS ONE;2017-01-19
5. Neuronal overexpression of human VAPB slows motor impairment and neuromuscular denervation in a mouse model of ALS;Human Molecular Genetics;2016-08-29
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