Inhibition of glutamate receptors reduces the homocysteine-induced whole blood platelet aggregation but does not affect superoxide anion generation or platelet membrane fluidization
Author:
Affiliation:
1. Department of Haemostatic Disorders, Chair of Biomedical Sciences, Medical University of Lodz, Lodz, Poland
2. Department of Medical Biophysics, Faculty of Biology and Environmental Protection, University of Lodz, Lodz, Poland
Funder
National Center of Science
Medical University of Lodz
Publisher
Informa UK Limited
Subject
Hematology,General Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/09537104.2016.1204438
Reference50 articles.
1. Homocysteine and vitamin therapy in stroke prevention and treatment: a review.
2. Homocysteine decreases platelet NO level via protein kinase C activation
3. The l-arginine/NO pathway in the early phases of platelet stimulation by collagen
4. Homocysteine impaired endothelial function through compromised vascular endothelial growth factor/Akt/endothelial nitric oxide synthase signalling
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