F11R/JAM-A: why do platelets express a molecule which is also present in tight junctions?
Author:
Affiliation:
1. Department of Haemostasis and Haemostatic Disorders, Medical University of Lodz, Lodz, Poland
2. Department of Medicine, State University of New York, Downstate Medical Center, Brooklyn, NY, USA
Funder
National Science Centre
Publisher
Informa UK Limited
Subject
Hematology,General Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/09537104.2023.2214618
Reference75 articles.
1. Activation of human platelets by a stimulatory monoclonal antibody.
2. Junctional Adhesion Molecule, a Novel Member of the Immunoglobulin Superfamily That Distributes at Intercellular Junctions and Modulates Monocyte Transmigration
3. Identification and characterisation of human Junctional Adhesion Molecule (JAM)
4. Silencing of the F11R gene reveals a role for F11R/JAM-A in the migration of inflamed vascular smooth muscle cells and in atherosclerosis
5. Mechanisms of platelet activation by a stimulatory antibody: cross-linking of a novel platelet receptor for monoclonal antibody F11 with the FcγRII receptor
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