Vanadate-Induced Inhibition of BCR-Triggered Apoptosis is Coupled with Tyrosine Phosphorylation and Induction of G2M Growth Arrest in Ramos-BL B Cells-Reference-Cited by-同舟云学术

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Vanadate-Induced Inhibition of BCR-Triggered Apoptosis is Coupled with Tyrosine Phosphorylation and Induction of G2M Growth Arrest in Ramos-BL B Cells

Published:2007-01 Issue:3 Volume:36 Page:293-306
ISSN:0882-0139
Container-title:Immunological Investigations
language:en
Short-container-title:Immunological Investigations

Author:

Hristov Kroum K.,Knox Kirstine A.,Mitev Vanio I.

Publisher

Informa UK Limited

Subject

General Medicine,Immunology

Link

http://www.tandfonline.com/doi/pdf/10.1080/08820130601069814

Reference47 articles.

1. Ligation of CD40 rescues Ramos-Burkitt lymphoma B cells from calcium ionophore- and antigen receptor-triggered apoptosis by inhibiting activation of the cysteine protease CPP32/Yama and cleavage of its substrate PARP

2. Protein tyrosine phosphatases: the quest for negative regulators of insulin action

3. Phosphorylation and identification of a major tyrosine phosphorylation site in protein tyrosine phosphatase 1C.

4. Critical role for mitochondria in B cell receptor‐mediated apoptosis

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Evaluation of immunotoxicity of sodium metavanadate following drinking water exposure in female B6C3F1/N mice in a 28‐day study;Journal of Applied Toxicology;2023-07-05

2. Regulation of cell adhesion to galectins by glycosylation: A new concept in lymphoma cell adhesion;Advances in Cancer Biology - Metastasis;2021-12

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