Zebrafish model of KRAS-initiated pancreatic endocrine tumor-Reference-Cited by-同舟云学术

Zebrafish model of KRAS-initiated pancreatic endocrine tumor

Published:2019-05-04 Issue:3 Volume:23 Page:209-218
ISSN:1976-8354
Container-title:Animal Cells and Systems
language:en
Short-container-title:Animal Cells and Systems

Author:

Oh Sekyung¹²,Park Joon Tae³^ORCID

Affiliation:

1. Department of Medicine, Catholic Kwandong University College of Medicine, Gangneung, Republic of Korea

2. Institute for Biomedical Research, Catholic Kwandong University International St. Mary’s Hospital, Catholic Kwandong University, Incheon, Republic of Korea

3. Division of Life Sciences, Incheon National University, Incheon, Republic of Korea

Funder

Incheon National University research grant

Publisher

Informa UK Limited

Subject

General Biochemistry, Genetics and Molecular Biology,Animal Science and Zoology

Link

https://tandfonline.com/doi/pdf/10.1080/19768354.2019.1610058

Reference20 articles.

1. Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma

2. In vivo cell biology: following the zebrafish trend

3. Three-Dimensional Collagen I Promotes Gemcitabine Resistance in Pancreatic Cancer through MT1-MMP–Mediated Expression of HMGA2

4. Chronic Pancreatitis Is Essential for Induction of Pancreatic Ductal Adenocarcinoma by K-Ras Oncogenes in Adult Mice

5. Suppression of Ptf1a Activity Induces Acinar-to-Endocrine Conversion

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