GANT61 suppresses cell survival, invasion and epithelial-mesenchymal transition through inactivating AKT/mTOR and JAK/STAT3 pathways in anaplastic thyroid carcinoma-Reference-Cited by-同舟云学术

GANT61 suppresses cell survival, invasion and epithelial-mesenchymal transition through inactivating AKT/mTOR and JAK/STAT3 pathways in anaplastic thyroid carcinoma

Published:2022-05-01 Issue:1 Volume:23 Page:369-377
ISSN:1538-4047
Container-title:Cancer Biology & Therapy
language:en
Short-container-title:Cancer Biology & Therapy

Author:

Gao Haoji¹,Wang Weige²,Li Qinyu¹^ORCID

Affiliation:

1. Department of General Surgery, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China

2. Medical Department, RIGEN Biotechnology Co., Ltd, Shanghai, China

Funder

funding

Publisher

Informa UK Limited

Subject

Cancer Research,Pharmacology,Oncology,Molecular Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384047.2022.2051158

Reference35 articles.

1. Anaplastic thyroid carcinoma: from clinicopathology to genetics and advanced therapies

2. American Thyroid Association Guidelines for Management of Patients with Anaplastic Thyroid Cancer

3. Patterns of Treatment Failure in Anaplastic Thyroid Carcinoma

4. Novel treatments for anaplastic thyroid carcinoma

5. Dabrafenib and Trametinib Treatment in Patients With Locally Advanced or Metastatic BRAF V600–Mutant Anaplastic Thyroid Cancer

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