Lipopolysaccharide (LPS) stimulation of Pancreatic Ductal Adenocarcinoma (PDAC) and macrophages activates the NLRP3 inflammasome that influences the levels of pro-inflammatory cytokines in a co-culture model-Reference-Cited by-同舟云学术

Lipopolysaccharide (LPS) stimulation of Pancreatic Ductal Adenocarcinoma (PDAC) and macrophages activates the NLRP3 inflammasome that influences the levels of pro-inflammatory cytokines in a co-culture model

Published:2023-11-29 Issue:1 Volume:24 Page:
ISSN:1538-4047
Container-title:Cancer Biology & Therapy
language:en
Short-container-title:Cancer Biology & Therapy

Author:

Sivam Hariss G Paremes¹^ORCID,Chin Beek Yoke²^ORCID,Gan Sook Yee³^ORCID,Ng Jia Hao⁴^ORCID,Gwenhure Agnes⁴^ORCID,Chan Elaine Wan Ling²^ORCID

Affiliation:

1. School of Health Sciences, International Medical University, Kuala Lumpur, Malaysia

2. Institute for Research, Development and Innovation, International Medical University, Kuala Lumpur, Malaysia

3. Department of Life Science, School of Pharmacy, International Medical University, Kuala Lumpur, Malaysia

4. School of Postgraduate Studies, International Medical University, Kuala Lumpur, Malaysia

Funder

International Medical University

Publisher

Informa UK Limited

Subject

Cancer Research,Pharmacology,Oncology,Molecular Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384047.2023.2284857

Reference59 articles.

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4. Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis

5. KrasG12D-Induced IKK2/β/NF-κB Activation by IL-1α and p62 Feedforward Loops Is Required for Development of Pancreatic Ductal Adenocarcinoma

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