Cyclin E overexpression confers resistance to trastuzumab through noncanonical phosphorylation of SMAD3 in HER2+ breast cancer
Author:
Affiliation:
1. Department of Biomedical Engineering, University of Michigan, Ann Arbor, MI, USA
2. Department of Surgery, Henry Ford Health System, Detroit, MI, USA
3. Department of Surgery, University of Michigan, Ann Arbor, MI, USA
Funder
NIH
A Sister’s Hope Foundation
Publisher
Informa UK Limited
Subject
Cancer Research,Pharmacology,Oncology,Molecular Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/15384047.2020.1818518
Reference67 articles.
1. Mechanisms of Disease: understanding resistance to HER2-targeted therapy in human breast cancer
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3. Trastuzumab — Mechanism of Action and Use in Clinical Practice
4. Use of Chemotherapy plus a Monoclonal Antibody against HER2 for Metastatic Breast Cancer That Overexpresses HER2
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