Crosstalk between PI3K/Akt and Wnt/β-catenin pathways promote colorectal cancer progression regardless of mutational status-Reference-Cited by-同舟云学术

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Crosstalk between PI3K/Akt and Wnt/β-catenin pathways promote colorectal cancer progression regardless of mutational status

Published:2022-08-09 Issue:1 Volume:23 Page:1-13
ISSN:1538-4047
Container-title:Cancer Biology & Therapy
language:en
Short-container-title:Cancer Biology & Therapy

Author:

Fleming-de-Moraes Cassio Dejair¹,Rocha Murilo Ramos¹^ORCID,Tessmann Josiane Weber¹,de Araujo Wallace Martins¹²,Morgado-Diaz Jose Andres¹^ORCID

Affiliation:

1. Cellular and Molecular Oncobiology Program, Cellular Dynamic and Structure Group, Instituto Nacional de Cancer – INCA, Rio de Janeiro, Brazil

2. Institute of Biological and Health Sciences, Federal Rural University of Rio de Janeiro, Rio de Janeiro, Brazil

Publisher

Informa UK Limited

Subject

Cancer Research,Pharmacology,Oncology,Molecular Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384047.2022.2108690

Reference47 articles.

1. Global cancer statistics 2018: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries

2. Pulmonary metastasis in newly diagnosed colon-rectal cancer: a population-based nomogram study

3. Wnt/β-Catenin Signaling, Disease, and Emerging Therapeutic Modalities

4. Oncogenic β-catenin and PIK3CA instruct network states and cancer phenotypes in intestinal organoids

5. Prognostic role of tumor PIK3CA mutation in colorectal cancer: a systematic review and meta-analysis

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