Defective autophagy in vascular smooth muscle cells enhances cell death and atherosclerosis-Reference-Cited by-同舟云学术

Defective autophagy in vascular smooth muscle cells enhances cell death and atherosclerosis

Published:2018-08-10 Issue:11 Volume:14 Page:1991-2006
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Osonoi Yusuke¹²,Mita Tomoya¹²³,Azuma Kosuke¹,Nakajima Kenichi¹,Masuyama Atsushi¹,Goto Hiromasa¹,Nishida Yuya¹,Miyatsuka Takeshi¹³,Fujitani Yoshio¹⁴,Koike Masato⁵^ORCID,Mitsumata Masako⁶,Watada Hirotaka¹²³

Affiliation:

1. Department of Metabolism & Endocrinology, Juntendo University Graduate School of Medicine , 2-1-1 Hongo, Bunkyo-ku, Tokyo 113-8421, Japan

2. Center for Identification of Diabetic Therapeutic Targets, Juntendo University Graduate School of Medicine , 2-1-1 Hongo, Bunkyo-ku, Tokyo 113-8421, Japan

3. Center for Therapeutic Innovations in Diabetes, Juntendo University Graduate School of Medicine , 2-1-1 Hongo, Bunkyo-ku, Tokyo 113-8421, Japan

4. Laboratory of Developmental Biology and Metabolism, Institute for Molecular and Cellular Regulation, Gunma University , Maebashi, Japan

5. Department of Cell Biology and Neuroscience, Juntendo University Graduate School of Medicine , Tokyo, Japan

6. Division of Cardiology, Department of Medicine, Nihon University School of Medicine , Tokyo, Japan

Funder

Ministry of Education, Sports and Culture of Japan

Japan Agency for Medical Research and Development

Juntendo University Young Investigator Joint Project Award 2015

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2018.1501132

Reference43 articles.

1. Autophagy: from phenomenology to molecular understanding in less than a decade

2. Loss of autophagy in the central nervous system causes neurodegeneration in mice

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4. The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stress

5. Autophagy Is Important in Islet Homeostasis and Compensatory Increase of Beta Cell Mass in Response to High-Fat Diet

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