LGALS3 (galectin 3) mediates an unconventional secretion of SNCA/α-synuclein in response to lysosomal membrane damage by the autophagic-lysosomal pathway in human midbrain dopamine neurons-Reference-Cited by-同舟云学术

LGALS3 (galectin 3) mediates an unconventional secretion of SNCA/α-synuclein in response to lysosomal membrane damage by the autophagic-lysosomal pathway in human midbrain dopamine neurons

Published:2021-10-06 Issue: Volume: Page:1-29
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Burbidge Kevin¹,Rademacher David J.²,Mattick Jessica³,Zack Stephanie³,Grillini Andrea⁴,Bousset Luc⁵,Kwon Ochan⁴,Kubicki Konrad⁴,Simon Alexander⁴,Melki Ronald⁵,Campbell Edward M.¹²

Affiliation:

1. Graduate Program in Neuroscience, Stritch School of Medicine, Loyola University Chicago, Maywood, Illinois, USA

2. Core Imaging Facility and Department of Microbiology and Immunology, Loyola University of Chicago, Maywood, Illinois, USA

3. Department of Microbiology and Immunology, Stritch School of Medicine, Loyola University, Chicago, Maywood, Illinois, USA

4. Stritch School of Medicine, Loyola University Chicago, Maywood, Illinois, USA

5. Institut Francois Jacob (Mircen), Cea and Laboratory of Neurodegenerative Diseases, Cnrs, Fontenay-Aux-Roses Cedex, France

Funder

Michael J. Fox Foundation for Parkinson’s Research

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2021.1967615

Reference127 articles.

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4. Cell-to-Cell Transmission of α-Synuclein Aggregates

5. Exosomal cell-to-cell transmission of alpha synuclein oligomers

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