A NOVEL NOX/PHOX-CD38-NAADP-TFEB AXIS IMPORTANT FOR MACROPHAGE ACTIVATION DURING BACTERIAL PHAGOCYTOSIS-Reference-Cited by-同舟云学术

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A NOVEL NOX/PHOX-CD38-NAADP-TFEB AXIS IMPORTANT FOR MACROPHAGE ACTIVATION DURING BACTERIAL PHAGOCYTOSIS

Published:2021-04-13 Issue: Volume: Page:1-18
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Najibi Mehran¹²,Honwad Havisha H.¹,Moreau Joseph A.¹,Becker Stephanie M.¹^ORCID,Irazoqui Javier E.¹^ORCID

Affiliation:

1. Department of Microbiology and Physiological Systems and Program in Innate Immunity, University of Massachusetts Medical School, Worcester, USA

2. Present Address: Department of Pathology, The Warren Alpert Medical School of Brown University, Providence

Funder

National Institute of General Medical Sciences

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2021.1911548

Reference82 articles.

1. MECHANISMS OF PHAGOCYTOSIS IN MACROPHAGES

2. Transcriptional regulation of macrophage polarization: enabling diversity with identity

3. Transcriptional control of the inflammatory response

4. Innate Host Defense Requires TFEB-Mediated Transcription of Cytoprotective and Antimicrobial Genes

5. An Evolutionarily Conserved PLC-PKD-TFEB Pathway for Host Defense

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1. TFEB-Mediated Pro-inflammatory Response in Murine Macrophages Induced by Acute Alpha7 Nicotinic Receptor Activation;2024-01-30

2. Salmonella actively modulates TFEB in murine macrophages in a growth-phase and time-dependent manner;Microbiology Spectrum;2024-01-11

3. SMURF1 controls the PPP3/calcineurin complex and TFEB at a regulatory node for lysosomal biogenesis;Autophagy;2023-11

4. Dissecting the multifaced function of transcription factor EB (TFEB) in human diseases: From molecular mechanism to pharmacological modulation;Biochemical Pharmacology;2023-09

5. A bacterial autotransporter impairs innate immune responses by targeting the transcription factor TFE3;Nature Communications;2023-04-11

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