Proteasome dysfunction induces excessive proteome instability and loss of mitostasis that can be mitigated by enhancing mitochondrial fusion or autophagy-Reference-Cited by-同舟云学术

Proteasome dysfunction induces excessive proteome instability and loss of mitostasis that can be mitigated by enhancing mitochondrial fusion or autophagy

Published:2019-04-19 Issue:10 Volume:15 Page:1757-1773
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Tsakiri Eleni N.¹,Gumeni Sentiljana¹,Vougas Konstantinos²,Pendin Diana³^ORCID,Papassideri Issidora¹,Daga Andrea⁴^ORCID,Gorgoulis Vassilis²⁵⁶,Juhász Gábor⁷^ORCID,Scorrano Luca⁸,Trougakos Ioannis P.¹

Affiliation:

1. Department of Cell Biology and Biophysics, Faculty of Biology, National and Kapodistrian University of Athens, Athens, Greece

2. Genomics and Proteomics Research Units, Center of Basic Research II, Biomedical Research Foundation, Academy of Athens, Athens, Greece

3. Department of Biomedical Sciences, University of Padova, Padova, Italy

4. Laboratory of Molecular Biology, Scientific Institute, IRCCS E. Medea, Lecco, Italy

5. Department of Histology and Embryology, School of Medicine, National and Kapodistrian University of Athens, Athens, Greece

6. Faculty of Biology, Medicine and Health, University of Manchester, Manchester, UK

7. Department of Anatomy, Cell and Developmental Biology, Eötvös Loránd University, Budapest, Hungary and Biological Research Centre, Hungarian Academy of Sciences, Szeged, Hungary

8. Dulbecco-Telethon Institute, Venetian Institute of Molecular Medicine and Department of Biology, University of Padua, Padova, Italy

Funder

General Secretariat for Research and Technology

Horizon 2020 Framework Programme

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://tandfonline.com/doi/pdf/10.1080/15548627.2019.1596477

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1. Non-enzymatic post-translational protein modifications and proteostasis network deregulation in carcinogenesis