SQSTM1/p62-mediated autophagy compensates for loss of proteasome polyubiquitin recruiting capacity-Reference-Cited by-同舟云学术

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SQSTM1/p62-mediated autophagy compensates for loss of proteasome polyubiquitin recruiting capacity

Published:2017-08-25 Issue:10 Volume:13 Page:1697-1708
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Demishtein Alik¹,Fraiberg Milana¹,Berko Dikla²,Tirosh Boaz³,Elazar Zvulun¹,Navon Ami²

Affiliation:

1. Department of Biomolecular Sciences, The Weizmann Institute of Science, Rehovot, Israel

2. Department of Biological Regulation, The Weizmann Institute of Science, Rehovot, Israel

3. Institute for Drug Research, The School of Pharmacy, Faculty of Medicine, The Hebrew University of Jerusalem, Jerusalem, Israel

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2017.1356549

Reference48 articles.

1. Autophagy: links with the proteasome

2. Autophagy and the ubiquitin-proteasome system: Collaborators in neuroprotection

3. Ubiquitination and selective autophagy

4. Getting to First Base in Proteasome Assembly

5. The multiubiquitin-chain-binding protein Mcb1 is a component of the 26S proteasome in Saccharomyces cerevisiae and plays a nonessential, substrate-specific role in protein turnover

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