Loss of ULK1 increases RPS6KB1-NCOR1 repression of NR1H/LXR-mediated Scd1 transcription and augments lipotoxicity in hepatic cells-Reference-Cited by-同舟云学术

Loss of ULK1 increases RPS6KB1-NCOR1 repression of NR1H/LXR-mediated Scd1 transcription and augments lipotoxicity in hepatic cells

Published:2017-01-02 Issue:1 Volume:13 Page:169-186
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Sinha Rohit Anthony¹,Singh Brijesh K.¹,Zhou Jin¹,Xie Sherwin¹,Farah Benjamin L.¹,Lesmana Ronny¹²,Ohba Kenji¹,Tripathi Madhulika³,Ghosh Sujoy¹,Hollenberg Anthony N.⁴,Yen Paul M.¹

Affiliation:

1. Laboratory of Hormonal Regulation, Cardiovascular and Metabolic Disorders Program, Duke-NUS Graduate Medical School, Singapore

2. Department of Physiology, Universitas Padjadjaran, Bandung, Indonesia

3. Stroke Trial Unit, National Neuroscience Institute Singapore, Singapore

4. Division of Endocrinology, Diabetes and Metabolism, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, MA USA

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://tandfonline.com/doi/pdf/10.1080/15548627.2016.1235123

Reference80 articles.

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4. Hepatic lipotoxicity and the pathogenesis of nonalcoholic steatohepatitis: The central role of nontriglyceride fatty acid metabolites

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