Pancreas-specific SNAP23 depletion prevents pancreatitis by attenuating pathological basolateral exocytosis and formation of trypsin-activating autolysosomes-Reference-Cited by-同舟云学术

Pancreas-specific SNAP23 depletion prevents pancreatitis by attenuating pathological basolateral exocytosis and formation of trypsin-activating autolysosomes

Published:2020-12-07 Issue:10 Volume:17 Page:3068-3081
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Dolai Subhankar¹,Takahashi Toshimasa²^ORCID,Qin Tairan²,Liang Tao²,Xie Li²,Kang Fei²^ORCID,Miao Yi-Fan²,Xie Huanli²,Kang Youhou²,Manuel Justin¹,Winter Erin¹,Roche Paul A.³,Cattral Mark S.¹,Gaisano Herbert Y.¹²

Affiliation:

1. Toronto General Hospital Research Institute, University Health Network, Toronto, Ontario, Canada

2. Department of Medicine, University of Toronto, Toronto, Ontario, Canada

3. Center for Cancer Research, National Cancer Institute, Bethesda, Maryland, USA

Funder

grant from Canadian Institute of Health Research

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2020.1852725

Reference59 articles.

1. Acute Pancreatitis: A Multifaceted Set of Organelle and Cellular Interactions

2. Recent Insights Into the Pathogenic Mechanism of Pancreatitis

3. Stimulus-secretion Coupling in Pancreatic Acinar Cells

4. Regulation of Physiologic and Pathologic Exocytosis in Pancreatic Acinar Cells

5. Basal autophagy maintains pancreatic acinar cell homeostasis and protein synthesis and prevents ER stress

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