PTPN9-mediated dephosphorylation of VTI1B promotes ATG16L1 precursor fusion and autophagosome formation-Reference-Cited by-同舟云学术

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PTPN9-mediated dephosphorylation of VTI1B promotes ATG16L1 precursor fusion and autophagosome formation

Published:2020-10-28 Issue:10 Volume:17 Page:2750-2765
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Chou He-Yen¹,Lee Yi-Tang¹²,Lin Yuchieh Jay¹²³,Wen Jung-Kun¹,Peng Wen-Hsin¹,Hsieh Pei-Lien¹,Lin Shu-Yu¹,Hung Chin-Chun¹,Chen Guang-Chao¹²³^ORCID

Affiliation:

1. Institute of Biological Chemistry, Academia Sinica, Taipei, Taiwan

2. Institute of Biochemical Sciences, College of Life Science, National Taiwan University, Taipei, Taiwan

3. Chemical Biology and Molecular Biophysics, Taiwan International Graduate Program, Academia Sinica, Taipei, Taiwan

Funder

Academia Sinica

Ministry of Science and Technology, Taiwan

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2020.1838117

Reference56 articles.

1. Autophagy: process and function

2. Initial Steps in Mammalian Autophagosome Biogenesis

3. ER exit sites are physical and functional core autophagosome biogenesis components

4. The ER–Golgi intermediate compartment is a key membrane source for the LC3 lipidation step of autophagosome biogenesis

5. AP1 is essential for generation of autophagosomes from trans-Golgi network

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