SMAD3 promotes autophagy dysregulation by triggering lysosome depletion in tubular epithelial cells in diabetic nephropathy-Reference-Cited by-同舟云学术

SMAD3 promotes autophagy dysregulation by triggering lysosome depletion in tubular epithelial cells in diabetic nephropathy

Published:2020-10-12 Issue:9 Volume:17 Page:2325-2344
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Yang Chen¹^ORCID,Chen Xiao-Cui¹,Li Zhi-Hang¹,Wu Hong-Luan¹,Jing Kai-Peng¹,Huang Xiao-Ru²,Ye Lin¹,Wei Biao²,Lan Hui-Yao²^ORCID,Liu Hua-Feng¹

Affiliation:

1. Key Laboratory of Prevention and Management of Chronic Kidney Disease of Zhanjiang City, Institute of Nephrology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, Guangdong, China

2. Department of Medicine & Therapeutics and Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Hong Kong SAR, China

Funder

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Research Grants Council of Hong Kong, University Grants Committee

Health and Medical Research Fund of Hong Kong

Guangdong Science and Technology Department

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2020.1824694

Reference61 articles.

1. Autophagy-Lysosome Pathway in Renal Tubular Epithelial Cells Is Disrupted by Advanced Glycation End Products in Diabetic Nephropathy

2. Urinary biomarkers for early diabetic nephropathy in type 2 diabetic patients

3. Metabonomics revealed xanthine oxidase-induced oxidative stress and inflammation in the pathogenesis of diabetic nephropathy

4. Twenty years after ACEIs and ARBs: emerging treatment strategies for diabetic nephropathy

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