Suppression of autophagy and HCK signaling promotes PTGS2high FCGR3− NK cell differentiation triggered by ectopic endometrial stromal cells-Reference-Cited by-同舟云学术

Suppression of autophagy and HCK signaling promotes PTGS2high FCGR3− NK cell differentiation triggered by ectopic endometrial stromal cells

Published:2018-07-20 Issue:8 Volume:14 Page:1376-1397
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Mei Jie¹²,Zhou Wen-Jie¹,Zhu Xiao-Yong¹³,Lu Han¹,Wu Ke¹,Yang Hui-Li¹,Fu Qiang⁴,Wei Chun-Yan¹,Chang Kai-Kai²,Jin Li-Ping⁵,Wang Jian¹,Wang Yong-Ming⁶,Li Da-Jin¹,Li Ming-Qing¹

Affiliation:

1. Laboratory for Reproductive Immunology, Key Laboratory of Reproduction Regulation of NPFPC, SIPPR, IRD, Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, People’s Republic of China

2. Reproductive Medicine Center, Department of Obstetrics and Gynecology, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medicine School, Nanjing, People’s Republic of China

3. Department of Gynecology, Hospital of Obstetrics and Gynecology, Shanghai Medical School, Fudan University, Shanghai, People’s Republic of China

4. Department of Immunology, Binzhou Medical College, Yantai, People’s Republic of China

5. Clinical and Translational Research Center, Shanghai First Maternity and Infant Hospital, Tongji University School of Medicine, Shanghai, People’s Republic of China

6. State Key Laboratory of Genetic Engineering, Collaborative Innovation Center for Genetics and Development, School of Life Sciences, Fudan University, Shanghai, People’s Republic of China

Funder

National Natural Science Foundation of China

National Basic Research Program of China

Shanghai Rising-Star Program

Development Fund of Shanghai Talents

Program of Shanghai Outstanding Academic Leader

Program for Zhuoxue of Fudan University

Nationan Science of Jiangsu Province

Fundamental Research Funds for the Central Universities

Program for Shanghai leaders

Oriented Project of Science and Technology Innovation from Key Lab. of Reproduction Regulation of NPFPC

Publisher