Fingolimod (FTY720) reduces viability and survival and increases histone H3 acetylation in medulloblastoma cells-Reference-Cited by-同舟云学术

Fingolimod (FTY720) reduces viability and survival and increases histone H3 acetylation in medulloblastoma cells

Published:2019-12-11 Issue:2 Volume:37 Page:170-175
ISSN:0888-0018
Container-title:Pediatric Hematology and Oncology
language:en
Short-container-title:Pediatric Hematology and Oncology

Author:

Perla Alexandre S.¹²,Fratini Lívia¹²,Cardoso Paula S.¹²,de Farias Caroline Brunetto¹³,da Cunha Jaeger Mariane¹³,Roesler Rafael¹²^ORCID

Affiliation:

1. Cancer and Neurobiology Laboratory, Experimental Research Center, Clinical Hospital (CPE-HCPA), Federal University of Rio Grande Do Sul, Porto Alegre, Brazil;

2. Department of Pharmacology, Institute for Basic Health Sciences, Federal University of Rio Grande Do Sul, Porto Alegre, Brazil;

3. Children’s Cancer Institute, Porto Alegre, Brazil

Publisher

Informa UK Limited

Subject

Oncology,Hematology,Pediatrics, Perinatology and Child Health

Link

https://www.tandfonline.com/doi/pdf/10.1080/08880018.2019.1699213

Reference19 articles.

1. Molecular subgroups of medulloblastoma: the current consensus

2. Oral Fingolimod (FTY720) for Relapsing Multiple Sclerosis

3. Induction of brain tumor stem cell apoptosis by FTY720: a potential therapeutic agent for glioblastoma

4. FTY720 reduces migration and invasion of human glioblastoma cell lines via inhibiting the PI3K/AKT/mTOR/p70S6K signaling pathway

5. The phosphorylated prodrug FTY720 is a histone deacetylase inhibitor that reactivates ERα expression and enhances hormonal therapy for breast cancer

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4. Vesicle Fusion as a Target Process for the Action of Sphingosine and Its Derived Drugs;International Journal of Molecular Sciences;2022-01-19

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