BCL11A Down-Regulation Induces γ-Globin in Human β-Thalassemia Major Erythroid Cells-Reference-Cited by-同舟云学术

BCL11A Down-Regulation Induces γ-Globin in Human β-Thalassemia Major Erythroid Cells

Published:2018-07-04 Issue:4 Volume:42 Page:225-230
ISSN:0363-0269
Container-title:Hemoglobin
language:en
Short-container-title:Hemoglobin

Author:

Li Jing¹,Lai Yongrong¹,Shi Lingling¹

Affiliation:

1. Department of Hematology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region, People’s Republic of China

Funder

National Natural Science Foundation of China

Research Fund for the Doctoral Program of Higher Education of China

Foundation of Department for Science and Technology of Guangxi Zhuang Autonomous Region

Publisher

Informa UK Limited

Subject

Biochemistry, medical,Clinical Biochemistry,Genetics(clinical),Hematology

Link

https://www.tandfonline.com/doi/pdf/10.1080/03630269.2018.1515774

Reference37 articles.

1. Beta-thalassemia

2. New Strategies in the Treatment of the Thalassemias

3. 4 Pathophysiology of thalassaemia

4. Absence of Haemoglobin A in an Individual Simultaneously Heterozygous in the Genes for Hereditary Persistence of Foetal Haemoglobin and β-Thalassemia°

5. Regulation of the β-and δ-Hemoglobin Genes

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