MYB3R-SCL28-SMR module with a role in cell size control negatively regulates G2 progression in <i>Arabidopsis</i>-Reference-Cited by-同舟云学术

MYB3R-SCL28-SMR module with a role in cell size control negatively regulates G2 progression in Arabidopsis

Published:2022-12-28 Issue: Volume: Page:1-5
ISSN:1559-2324
Container-title:Plant Signaling & Behavior
language:en
Short-container-title:Plant Signaling & Behavior

Author:

Takatsuka Hirotomo¹,Nomoto Yuji¹,Yamada Kesuke¹,Mineta Keito¹,Breuer Christian²,Ishida Takashi²³,Yamagami Ayumi⁴⁵,Sugimoto Keiko²⁶,Nakano Takeshi⁴⁵⁷,Ito Masaki¹

Affiliation:

1. School of Biological Science and Technology, College of Science and Engineering, Kanazawa University, Kakuma-machi, Kanazawa, 920-1192, Japan

2. RIKEN Center for Sustainable Resource Science, Yokohama, 230-0045, Japan

3. Faculty of Advanced Science and Technology, Kumamoto University, Kumamoto, 860-8555, Japan

4. Graduate School of Biostudies, Kyoto University, Kyoto, Japan

5. Gene Discovery Research Group, RIKEN Center for Sustainable Resource Science, Tsukuba, Japan

6. Department of Biological Sciences, Graduate School of Science, the University of Tokyo, 7-3-1 Hongo, Bunkyo-ku, Tokyo 113-0033, Japan

7. School of Engineering and Applied Sciences, National University of Mongolia, Ulaanbaatar, Mongolia

Funder

The Japan Society for the Promotion of Science KAKENHI

Japan Science and Technology Agency

MEXT Leading Initiative for Excellent Young Researchers, and Kanazawa University JIKOCHOKOKU project to H.T

Publisher

Informa UK Limited

Subject

Plant Science

Link

https://www.tandfonline.com/doi/pdf/10.1080/15592324.2022.2153209

Reference18 articles.

1. Endoreplication — a means to an end in cell growth and stress response

2. Programmed induction of endoreduplication by DNA double-strand breaks in Arabidopsis

3. A hierarchical transcriptional network activates specific CDK inhibitors that regulate G2 to control cell size and number in Arabidopsis

4. G2/M-Phase–Specific Transcription during the Plant Cell Cycle Is Mediated by c-Myb–Like Transcription Factors

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