N 6 -methyladenosine (M 6 A) in fetal offspring modifies mitochondrial gene expression following gestational nano-TiO 2 inhalation exposure-Reference-Cited by-同舟云学术

N ⁶ -methyladenosine (M ⁶ A) in fetal offspring modifies mitochondrial gene expression following gestational nano-TiO ₂ inhalation exposure

Published:2023-11-26 Issue:10 Volume:17 Page:651-668
ISSN:1743-5390
Container-title:Nanotoxicology
language:en
Short-container-title:Nanotoxicology

Author:

Kunovac Amina¹²³,Hathaway Quincy A.¹⁴^ORCID,Thapa Dharendra¹²,Durr Andrya J.¹²,Taylor Andrew D.¹²,Rizwan Saira¹²,Sharif Daud⁵,Valentine Stephen J.⁵,Hollander John M.¹²

Affiliation:

1. Division of Exercise Physiology, West Virginia University School of Medicine, Morgantown, WV, USA

2. Mitochondria, Metabolism & Bioenergetics Working Group, West Virginia University School of Medicine, Morgantown, WV, USA

3. Center for Inhalation Toxicology (iTOX), West Virginia University School of Medicine, Morgantown, WV, USA

4. Department of Medical Education, West Virginia University School of Medicine, Morgantown, WV, USA

5. Department of Chemistry, West Virginia University, Morgantown, WV, USA

Funder

American Heart Association

National Heart, Lung, and Blood Institute

National Institute of Environmental Health Sciences

Community Foundation for the Ohio Valley Whipkey Trust

Publisher

Informa UK Limited

Link

https://www.tandfonline.com/doi/pdf/10.1080/17435390.2023.2293144

Reference69 articles.

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4. Proteomic alterations of distinct mitochondrial subpopulations in the type 1 diabetic heart: contribution of protein import dysfunction

5. Changes in m6A RNA methylation contribute to heart failure progression by modulating translation