USP7-stabilised HIPK2 promotes high glucose-induced endothelial cell dysfunctions to accelerate diabetic foot ulcers-Reference-Cited by-同舟云学术

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USP7-stabilised HIPK2 promotes high glucose-induced endothelial cell dysfunctions to accelerate diabetic foot ulcers

Published:2024-07-27 Issue: Volume: Page:1-8
ISSN:1381-3455
Container-title:Archives of Physiology and Biochemistry
language:en
Short-container-title:Archives of Physiology and Biochemistry

Author:

Huang Huimin¹,Huang Yangyong²

Affiliation:

1. Burn & Plastic & Wound Surgery Department, The First College of Clinical Medical Science, China Three Gorges University, Yichang, China

2. Department of Colorectal Surgery, The Affiliated TCM Hospital of Guangzhou Medical University, Guangzhou, China

Publisher

Informa UK Limited

Link

https://www.tandfonline.com/doi/pdf/10.1080/13813455.2024.2376815

Reference33 articles.

1. Diabetic Foot Ulcers and Their Recurrence

2. The diabetic foot: Pathophysiology, evaluation, and treatment

3. Diabetic foot management: multidisciplinary approach for advanced lesion rescue

4. HIPK2 suppresses tumor growth and progression of hepatocellular carcinoma through promoting the degradation of HIF-1α

5. Deubiquitination of FBP1 by USP7 blocks FBP1–DNMT1 interaction and decreases the sensitivity of pancreatic cancer cells to PARP inhibitors

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