High glucose induces inflammatory responses in HepG2 cells via the oxidative stress-mediated activation of NF-κB, and MAPK pathways in HepG2 cells-Reference-Cited by-同舟云学术

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High glucose induces inflammatory responses in HepG2 cells via the oxidative stress-mediated activation of NF-κB, and MAPK pathways in HepG2 cells

Published:2018-01-24 Issue:5 Volume:124 Page:468-474
ISSN:1381-3455
Container-title:Archives of Physiology and Biochemistry
language:en
Short-container-title:Archives of Physiology and Biochemistry

Author:

Panahi Ghodratollah¹,Pasalar Parvin¹,Zare Mina²,Rizzuto Rosario³,Meshkani Reza¹^ORCID

Affiliation:

1. Department of Biochemistry, Faculty of Medicine, Tehran University of Medical Sciences, Tehran, Iran;

2. Recombinant Protein Laboratory, Department of Biochemistry, Shiraz University of Medical Sciences, Shiraz, Iran;

3. Department of Biomedical Sciences, University of Padova, Padua, Italy

Funder

Tehran University of Medical Sciences and Health Services

Publisher

Informa UK Limited

Subject

Physiology (medical),Physiology,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/13813455.2018.1427764

Reference42 articles.

1. Diagnosis and Classification of Diabetes Mellitus

2. p38 MAPK-induced Nuclear Factor-κB Activity Is Required for Skeletal Muscle Differentiation: Role of Interleukin-6

3. Activation and Function of the MAPKs and Their Substrates, the MAPK-Activated Protein Kinases

4. Inflammation and the Metabolic Syndrome: The Tissue-Specific Functions of NF-κB

5. High glucose-induced proteome alterations in hepatocytes and its possible relevance to diabetic liver disease

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