Role of mitochondrial DNA in oxidative damage induced by sodium arsenite in human bronchial epithelial cells
Author:
Affiliation:
1. Department of Toxicology, School of Public Health, Jiangsu Key Laboratory of Preventive and Translational Medicine for Geriatric Diseases, Medical College of Soochow University, Suzhou, Jiangsu, People’s Republic of China
Publisher
Informa UK Limited
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
https://www.tandfonline.com/doi/pdf/10.1080/15287394.2019.1686108
Reference33 articles.
1. AP-1 activation attenuates the arsenite-induced apoptotic response in human bronchial epithelial cells by up-regulating HO-1 expression
2. Inflammation and Lung Cancer: Roles of Reactive Oxygen/Nitrogen Species
3. Mitochondrial Oxygen Consumption and Reactive Oxygen Species Production are Independently Modulated: Implications for Aging Studies
4. MOLECULAR ASPECTS OF ARSENIC STRESS
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3. Comparative study of two isothiazolinone biocides, 1,2-benzisothiazolin-3-one (BIT) and 4,5-dichloro-2-n-octyl-isothiazolin-3-one (DCOIT), on barrier function and mitochondrial bioenergetics using murine brain endothelial cell line (bEND.3);Journal of Toxicology and Environmental Health, Part A;2021-07-27
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