The protective mechanism underlying phenylethanoid glycosides (PHG) actions on synaptic plasticity in rat Alzheimer’s disease model induced by beta amyloid 1-42-Reference-Cited by-同舟云学术

The protective mechanism underlying phenylethanoid glycosides (PHG) actions on synaptic plasticity in rat Alzheimer’s disease model induced by beta amyloid 1-42

Published:2018-11-02 Issue:21 Volume:81 Page:1098-1107
ISSN:1528-7394
Container-title:Journal of Toxicology and Environmental Health, Part A
language:en
Short-container-title:Journal of Toxicology and Environmental Health, Part A

Author:

Jia Jian-xin¹,Yan Xu-sheng¹,Song Wei¹,Fang Xin¹,Cai Zhi-ping¹,Huo Dong-sheng¹,Wang He²,Yang Zhan-jun¹

Affiliation:

1. Department of Human Anatomy, Baotou Medical College, Inner Mongolia, China

2. School of Health Sciences, University of Newcastle, Newcastle, Australia

Funder

National Natural Science Foundation of China

Natural Science Foundation of Inner Mongolia Autonomous Region of China

Science and Technology Planning Project of Inner Mongolia Autonomous Region of China

Yangfan Project

Dengfeng Project

Publisher

Informa UK Limited

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15287394.2018.1501861

Reference35 articles.

1. Amyloid β oligomers induce Ca2+ dysregulation and neuronal death through activation of ionotropic glutamate receptors

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3. Prefrontal and hippocampal contributions to encoding and retrieval of spatial memory

4. The intersection of amyloid beta and tau in glutamatergic synaptic dysfunction and collapse in Alzheimer's disease

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