Trex2 responds to damaged replication forks in diverse ways
Author:
Affiliation:
1. Department of Molecular Medicine and Institute of Biotechnology, The Cancer Therapy Research Center, Sam, Ann Barshop Institute for Longevity and Aging Studies, University of Texas Health San Antonio, San Antonio, Texas, USA
Funder
National Cancer Institute
National Institute on Aging
Office of AIDS Research
Publisher
Informa UK Limited
Subject
Cancer Research,Molecular Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/23723556.2021.1881394
Reference10 articles.
1. TREX2 Exonuclease Causes Spontaneous Mutations and Stress-Induced Replication Fork Defects in Cells Expressing RAD51K133A
2. Two replication fork maintenance pathways fuse inverted repeats to rearrange chromosomes
3. Biochemical and cellular characteristics of the 3' -> 5' exonuclease TREX2
4. RAD51 Mutants Cause Replication Defects and Chromosomal Instability
5. The phenotype of FancB-mutant mouse embryonic stem cells
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