Persistent Acetylation of Histone H3 Lysine 56 Compromises the Activity of DNA Replication Origins-Reference-Cited by-同舟云学术

Persistent Acetylation of Histone H3 Lysine 56 Compromises the Activity of DNA Replication Origins

Published:2023-10-09 Issue:11 Volume:43 Page:566-595
ISSN:1098-5549
Container-title:Molecular and Cellular Biology
language:en
Short-container-title:Molecular and Cellular Biology

Author:

Tremblay Roch¹²,Mehrjoo Yosra¹²,Ahmed Oumaima¹²,Simoneau Antoine¹²,McQuaid Mary E.¹,Affar El Bachir¹³,Nislow Corey⁴,Giaever Guri⁴,Wurtele Hugo¹³^ORCID

Affiliation:

1. Maisonneuve-Rosemont Hospital Research Center, Montreal, Québec, Canada

2. Molecular Biology Program, Université de Montréal, Montreal, Québec, Canada

3. Department of Medicine, Université de Montréal, Montreal, Québec, Canada

4. Department of Pharmaceutical Sciences, University of British Columbia, Vancouver, British Columbia, Canada

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/10985549.2023.2259739

Reference82 articles.

1. Eukaryotic DNA replication control: Lock and load, then fire

2. Sld7, an Sld3-associated protein required for efficient chromosomal DNA replication in budding yeast

3. Limiting replication initiation factors execute the temporal programme of origin firing in budding yeast

4. The checkpoint response to replication stress

5. Mrc1 and Rad9 cooperate to regulate initiation and elongation of DNA replication in response to DNA damage

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