Distinctive alteration in the expression of autophagy genes in Drosophila models of amyloidopathy and tauopathy
Author:
Affiliation:
1. Department of Biology, College of Sciences, Shiraz University, Shiraz, Iran
2. Center for Molecular Protein Science, Lund University, Lund, Sweden
3. Department of Statistics, College of Sciences, Shiraz University, Shiraz, Iran
Funder
Shiraz University Research Council
Swedish Research Council grant
Publisher
Uppsala Medical Society
Subject
General Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/03009734.2020.1785063
Reference62 articles.
1. Alzheimer Disease–Associated Peptide, Amyloid β40, Inhibits Vascular Regeneration With Induction of Endothelial Autophagy
2. Naturally Secreted Amyloid-β Increases Mammalian Target of Rapamycin (mTOR) Activity via a PRAS40-mediated Mechanism
3. Soluble amyloid-beta buffering by plaques in Alzheimer disease dementia versus high-pathology controls
4. Is Tau Aggregation Toxic or Protective: A Sensible Question in the Absence of Sensitive Methods?
5. Lysosomal Activation Is a Compensatory Response Against Protein Accumulation and Associated Synaptopathogenesis—An Approach for Slowing Alzheimer Disease?
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