TXNIP deficiency attenuates renal fibrosis by modulating mTORC1/TFEB-mediated autophagy in diabetic kidney disease-Reference-Cited by-同舟云学术

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TXNIP deficiency attenuates renal fibrosis by modulating mTORC1/TFEB-mediated autophagy in diabetic kidney disease

Published:2024-04-14 Issue:1 Volume:46 Page:
ISSN:0886-022X
Container-title:Renal Failure
language:en
Short-container-title:Renal Failure

Author:

Du Yunxia¹²³,Wu Ming¹²³,Song Shan¹²³,Bian Yawei¹,Shi Yonghong¹²³

Affiliation:

1. Department of Pathology, Hebei Medical University, Shijiazhuang, China

2. Hebei Key Laboratory of Kidney Disease, Shijiazhuang, China

3. Center of Metabolic Diseases and Cancer Research, Institute of Medical and Health Science, Hebei Medical University, Shijiazhuang, China

Funder

the National Natural Science Foundation of China

the Natural Science Foundation of Hebei Province

Clinical Medicine Innovation Research Team Support Project of Hebei Medical University

Publisher

Informa UK Limited

Link

https://www.tandfonline.com/doi/pdf/10.1080/0886022X.2024.2338933

Reference50 articles.

1. Molecular mechanisms of diabetic kidney disease

2. Diabetes and Kidney Disease: Role of Oxidative Stress

3. VASH-1 Regulates Oxidative Stress and Fibrosis in Diabetic Kidney Disease via SIRT1/HIF1α and TGFβ1/Smad3 Signaling Pathways

4. NADPH Oxidases, Reactive Oxygen Species, and the Kidney: Friend and Foe

5. Hyperglycemia Promotes Oxidative Stress through Inhibition of Thioredoxin Function by Thioredoxin-interacting Protein

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