Inhibition of PID1/NYGGF4/PCLI1 gene expression highlights its role in the early events of the cell cycle in NIH3T3 fibroblasts-Reference-Cited by-同舟云学术

Inhibition of PID1/NYGGF4/PCLI1 gene expression highlights its role in the early events of the cell cycle in NIH3T3 fibroblasts

Published:2016-08-17 Issue:sup4 Volume:31 Page:45-53
ISSN:1475-6366
Container-title:Journal of Enzyme Inhibition and Medicinal Chemistry
language:en
Short-container-title:Journal of Enzyme Inhibition and Medicinal Chemistry

Author:

Monteleone Francesca¹,Vitale Monica¹²³,Caratù Ginevra¹²,D’Ambrosio Chiara³,Di Giovanni Stefano²,Gorrese Marisa¹,Napolitano Francesco¹,Romano Maria Fiammetta²,Del Vecchio Luigi¹²,Succoio Mariangela¹²,Scaloni Andrea³,Zambrano Nicola¹²

Affiliation:

1. CEINGE Biotecnologie Avanzate, Napoli, Italy,

2. Dipartimento di Medicina Molecolare e Biotecnologie Mediche, Università degli Studi di Napoli Federico II, Napoli, Italy, and

3. Proteomics & Mass Spectrometry Laboratory, ISPAAM – National Research Council, Naples, Italy

Publisher

Informa UK Limited

Subject

Drug Discovery,Pharmacology,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/14756366.2016.1217855

Reference26 articles.

1. Identification and characterization of NYGGF4, a novel gene containing a phosphotyrosine-binding (PTB) domain that stimulates 3T3-L1 preadipocytes proliferation

2. Identification of the Ligands of Protein Interaction Domains through a Functional Approach

3. Extensive proteomic screening identifies the obesity-related NYGGF4 protein as a novel LRP1-interactor, showing reduced expression in early Alzheimer's disease

4. Over-expression of NYGGF4 inhibits glucose transport in 3T3-L1 adipocytes via attenuated phosphorylation of IRS-1 and Akt

5. Over-expression of NYGGF4 (PID1) inhibits glucose transport in skeletal myotubes by blocking the IRS1/PI3K/AKT insulin pathway

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