Glutamine stimulates the S6K/4E-BP branch of insulin signalling pathway to mitigate human poly(Q) disorders in <i>Drosophila</i> disease models-Reference-Cited by-同舟云学术

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Glutamine stimulates the S6K/4E-BP branch of insulin signalling pathway to mitigate human poly(Q) disorders in Drosophila disease models

Published:2023-09-02 Issue: Volume: Page:1-12
ISSN:1028-415X
Container-title:Nutritional Neuroscience
language:en
Short-container-title:Nutritional Neuroscience

Author:

Tandon Shweta¹,Sarkar Surajit¹^ORCID

Affiliation:

1. Department of Genetics, University of Delhi South Campus, New Delhi, India

Funder

Department of Biotechnology, Government of India, New Delhi

University Grants Commission

Publisher

Informa UK Limited

Subject

Nutrition and Dietetics,General Medicine,General Neuroscience,Medicine (miscellaneous)

Link

https://www.tandfonline.com/doi/pdf/10.1080/1028415X.2023.2253028

Reference39 articles.

1. Polyglutamine expansion diseases: More than simple repeats

2. Polyglutamine diseases

3. Polyglutamine Repeats in Neurodegenerative Diseases

4. Tissue-Specific Upregulation of Drosophila Insulin Receptor (InR) Mitigates Poly(Q)-Mediated Neurotoxicity by Restoration of Cellular Transcription Machinery

5. The S6k/4E-BP mediated growth promoting sub-pathway of insulin signalling cascade is essential to restrict pathogenesis of poly(Q) disorders in Drosophila

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Polyglutamine disorders: Pathogenesis and potential drug interventions;Life Sciences;2024-05

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