The role of nitric oxide in paraquat-induced cytotoxicity in the human A549 lung carcinoma cell line
Author:
Publisher
Informa UK Limited
Subject
General Medicine,Biochemistry
Link
http://www.tandfonline.com/doi/pdf/10.1080/10715760100300181
Reference26 articles.
1. Superoxide- and singlet oxygen-catalyzed lipid peroxidation as a possible mechanism for paraquat (methyl viologen) toxicity
2. In vitro stimulation by paraquat of reactive oxygen-mediated lipid peroxidation in rat lung microsomes
3. Paraquat and NADPH-dependent lipid peroxidation in lung microsomes.
4. Nitric oxide as a mediator of oxidant lung injury due to paraquat.
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1. Paraquat toxicity is attenuated by 4-phenylbutyrate-induced phosphorylation of ERK2 via PI3K in A549 cells;Biochemical and Biophysical Research Communications;2018-09
2. Eukaryotic translation initiation factor 2 subunit α (eIF2α) inhibitor salubrinal attenuates paraquat-induced human lung epithelial-like A549 cell apoptosis by regulating the PERK-eIF2α signaling pathway;Toxicology in Vitro;2018-02
3. Sodium tauroursodeoxycholate prevents paraquat-induced cell death by suppressing endoplasmic reticulum stress responses in human lung epithelial A549 cells;Biochemical and Biophysical Research Communications;2013-03
4. Role of thioredoxin in lung disease;Pulmonary Pharmacology & Therapeutics;2012-04
5. Therapeutic hypothermia attenuates acute lung injury in paraquat intoxication in rats;Resuscitation;2011-04
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