Suppression of innate immune signaling molecule, MAVS, reduces radiation-induced bystander effect-Reference-Cited by-同舟云学术

Suppression of innate immune signaling molecule, MAVS, reduces radiation-induced bystander effect

Published:2020-08-20 Issue:1 Volume:97 Page:102-110
ISSN:0955-3002
Container-title:International Journal of Radiation Biology
language:en
Short-container-title:International Journal of Radiation Biology

Author:

Jia Rong¹²,Chen Yaxiong¹,Jia Cong³,Hu Burong¹²,Du Yarong¹³

Affiliation:

1. Institute of Modern Physics, Chinese Academy of Sciences, Key Laboratory of Space Radiobiology of Gansu Province & CAS Key Laboratory of Heavy Ion Radiation Biology and Medicine, Lanzhou, China

2. University of Chinese Academy of Sciences, Beijing, China

3. College of Life Science, Northwest Normal University, Lanzhou, China

Funder

National Natural Science Foundation of China

Science and Technology Research Project of Gansu Province

Publisher

Informa UK Limited

Subject

Radiology Nuclear Medicine and imaging,Radiological and Ultrasound Technology

Link

https://www.tandfonline.com/doi/pdf/10.1080/09553002.2020.1807642

Reference32 articles.

1. Dynamics of DNA Double-Strand Breaks Revealed by Clustering of Damaged Chromosome Domains

2. Ionizing radiation-induced metabolic oxidative stress and prolonged cell injury

3. Mitochondria in innate immune signaling

4. Reactive oxygen species induce virus-independent MAVS oligomerization in systemic lupus erythematosus

5. MAVS ubiquitination by the E3 ligase TRIM25 and degradation by the proteasome is involved in type I interferon production after activation of the antiviral RIG-I-like receptors

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