The intercellular communications mediating radiation-induced bystander effects and their relevance to environmental, occupational, and therapeutic exposures-Reference-Cited by-同舟云学术

The intercellular communications mediating radiation-induced bystander effects and their relevance to environmental, occupational, and therapeutic exposures

Published:2022-05-27 Issue: Volume: Page:1-19
ISSN:0955-3002
Container-title:International Journal of Radiation Biology
language:en
Short-container-title:International Journal of Radiation Biology

Author:

Buonanno Manuela¹^ORCID,Gonon Géraldine²^ORCID,Pandey Badri N.³^ORCID,Azzam Edouard I.⁴⁵^ORCID

Affiliation:

1. Center for Radiological Research, Columbia University Irving Medical Center, New York, NY, USA

2. Institut de Radioprotection et de Sûreté Nucléaire (IRSN), PSE-SANTE/SERAMED/LRAcc, Fontenay-aux-Roses, France

3. Radiation Biology and Health Sciences Division, Bhabha Atomic Research Centre, Mumbai, India

4. Radiobiology and Health Branch, Isotopes, Radiobiology & Environment Directorate (IRED), Canadian Nuclear Laboratories (CNL), Deep River, Canada

5. Department of Radiology, Rutgers New Jersey Medical School, Newark, NJ, USA

Funder

National Institutes of Health

National Aeronautics and Space Administration

US department of Energy Low Dose Radiation Research Program

New Jersey Commission on Cancer Research

Federal Nuclear Science and Technology Program at Canadian Nuclear Laboratories

National Cancer Institute

Publisher

Informa UK Limited

Subject

Radiology, Nuclear Medicine and imaging,Radiological and Ultrasound Technology

Link

https://www.tandfonline.com/doi/pdf/10.1080/09553002.2022.2078006

Reference225 articles.

1. Connexins in cancer: bridging the gap to the clinic

2. Enhancement of Peroxidase Release from Non-Malignant and Malignant Cells through Low-Dose Irradiation with Different Radiation Quality

3. Ionizing radiation affects the composition of the proteome of extracellular vesicles released by head-and-neck cancer cells in vitro

4. FasL-triggered death of Jurkat cells requires caspase 8-induced, ATP-dependent cross-talk between fas and the purinergic receptor P2X7

5. The non-targeted effects of radiation are perpetuated by exosomes

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