Upregulated IL-1 Receptor-associated Kinase 1 (IRAK1) in Systemic Lupus Erythematosus: IRAK1 Inhibition Represses Th17 Differentiation with Therapeutic Potential-Reference-Cited by-同舟云学术

Upregulated IL-1 Receptor-associated Kinase 1 (IRAK1) in Systemic Lupus Erythematosus: IRAK1 Inhibition Represses Th17 Differentiation with Therapeutic Potential

Published:2018-04-03 Issue:5 Volume:47 Page:468-483
ISSN:0882-0139
Container-title:Immunological Investigations
language:en
Short-container-title:Immunological Investigations

Author:

Zhou Zhou¹,Tian Zhiqiang²,Zhang Mengjie²,Zhang Yuxun³,Ni Bing²,Hao Fei¹

Affiliation:

1. Department of Dermatology, Southwest Hospital, Third Military Medical University, Chongqing, China

2. Institute of Immunology, Third Military Medical University, Chongqing, China

3. College of Liberal Arts and Sciences, University of lowa, lowa City, USA

Funder

National Natural Science Foundation of China

Publisher

Informa UK Limited

Subject

General Medicine,Immunology

Link

https://www.tandfonline.com/doi/pdf/10.1080/08820139.2018.1458105

Reference51 articles.

1. Translocation of the interleukin-1 receptor-associated kinase-1 (IRAK-1) into the nucleus

2. Cellular trafficking of the IL-1RI-associated kinase-1 requires intact kinase activity

3. Derivation of the sledai. A disease activity index for lupus patients

4. MicroRNA-146a in autoimmunity and innate immune responses

5. Decreased plasma IL22 levels, but not increased IL17 and IL23 levels, correlate with disease activity in patients with systemic lupus erythematosus

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