Further analysis of KIFAP3 gene in ALS patients from Switzerland and Sweden-Reference-Cited by-同舟云学术

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Further analysis of KIFAP3 gene in ALS patients from Switzerland and Sweden

Published:2017-01-31 Issue:3-4 Volume:18 Page:302-304
ISSN:2167-8421
Container-title:Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration
language:en
Short-container-title:Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration

Author:

Czell David¹,Sapp Peter C.²,Neuwirth Christoph³,Weber Markus³,Andersen Peter M.⁴,Brown Robert H.²

Affiliation:

1. Department of Neurology, Spital Linth, Uznach, Switzerland,

2. Department of Neurology, University of Massachusetts Medical School, Worcester, MA, USA,

3. Neuromuscular Unit, Cantonal Hospital St. Gallen, Switzerland, and

4. Department of Pharmacology and Clinical Neuroscience, Umeå University, Umeå, Sweden

Publisher

Informa UK Limited

Subject

Neurology (clinical),Neurology

Link

https://www.tandfonline.com/doi/pdf/10.1080/21678421.2017.1280509

Reference8 articles.

1. Credibility Analysis of Putative Disease-Causing Genes Using Bioinformatics

2. Targeted Exon Capture and Sequencing in Sporadic Amyotrophic Lateral Sclerosis

3. Variants in candidate ALS modifier genes linked to Cu/Zn superoxide dismutase do not explain divergent survival phenotypes

4. Reduced expression of the Kinesin-Associated Protein 3 (KIFAP3) gene increases survival in sporadic amyotrophic lateral sclerosis

5. Kinesin-associated protein 3 (KIFAP3) has no effect on survival in a population-based cohort of ALS patients

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. A human proteogenomic-cellular framework identifies KIF5A as a modulator of astrocyte process integrity with relevance to ALS;Communications Biology;2023-06-29

2. Genetic factors for survival in amyotrophic lateral sclerosis: an integrated approach combining a systematic review, pairwise and network meta-analysis;BMC Medicine;2022-06-27

3. Modified age-dependent expression of NaV1.6 in an ALS model correlates with motor cortex excitability alterations;Neurobiology of Disease;2019-10

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