Continuous overexpression of thioredoxin 1 enhances cancer development and does not extend maximum lifespan in male C57BL/6 mice-Reference-Cited by-同舟云学术

Continuous overexpression of thioredoxin 1 enhances cancer development and does not extend maximum lifespan in male C57BL/6 mice

Published:2018-01 Issue:1 Volume:8 Page:1533754
ISSN:2001-0001
Container-title:Pathobiology of Aging & Age-related Diseases
language:en
Short-container-title:Pathobiology of Aging & Age-related Diseases

Author:

Flores Lisa C.¹,Roman Madeline G.¹,Cunningham Geneva M.¹,Cheng Christie¹,Dube Sara¹,Allen Colton¹,Van Remmen Holly²,Hubbard Gene B.¹³,Saunders Thomas L.⁴,Ikeno Yuji¹³⁵

Affiliation:

1. Barshop Institute for Longevity and Aging Studies, The University of Texas Health Science Center at San Antonio, San Antonio, TX, USA

2. Aging and Metabolism Research Program, Oklahoma Medical Research Foundation, Oklahoma City, OK, USA

3. Department of Pathology, The University of Texas Health Science Center at San Antonio, San Antonio, TX, USA

4. Transgenic Animal Model Core, University of Michigan, Ann Arbor, MI, USA

5. Geriatric Research Education and Clinical Center (GRECC), Audie L. Murphy VA Hospital, South Texas Veterans Health Care System, San Antonio, TX, USA

Funder

NIH

Publisher

Informa UK Limited

Subject

Geriatrics and Gerontology,Aging,Histology

Link

https://www.tandfonline.com/doi/pdf/10.1080/20010001.2018.1533754

Reference46 articles.

1. ATL-derived factor (ADF), an IL-2 receptor/Tac inducer homologous to thioredoxin; possible involvement of dithiol-reduction in the IL-2 receptor induction.

2. Cloning and Expression of a Novel Mammalian Thioredoxin

3. Characterization of three isoforms of mammalian peroxiredoxin that reduce peroxides in the presence of thioredoxin

4. Isoforms of mammalian peroxiredoxin that reduce peroxides in presence of thioredoxin

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