The Case for Pro-γ-MSH as the Adrenal Growth Factor
Author:
Publisher
Informa UK Limited
Subject
Endocrinology,General Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1080/07435808409036500
Reference28 articles.
1. BIOASSAY OF ADRENOCORTICOTROPIC HORMONE1
2. THE ASSAY OF ADRENOCORTICOTROPHIC HORMONE BY THE ADRENAL ASCORBIC ACID-DEPLETION METHOD1
3. STRUCTURE OF β-CORTICOTROPIN: FINAL SEQUENCE STUDIES
4. Studies with Corticotropin. III. Determination of the Structure of β-Corticotropin1 and its Active Degradation Products
5. CORTICOSTEROID SECRETION IN THE ADRENAL VENOUS BLOOD OF THE HYPOPHYSECTOMIZED DOG AS AN ASSAY FOR ACTH1
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1. Peripheral administration of the N-terminal pro-opiomelanocortin fragment 1–28 to Pomc−/− mice reduces food intake and weight but does not affect adrenal growth or corticosterone production;Journal of Endocrinology;2006-08
2. Evidence against a role of human airway trypsin-like protease – the human analogue of the growth-promoting rat adrenal secretory protease – in adrenal tumourigenesis;European Journal of Endocrinology;2005-01
3. The Adrenal Secretory Serine Protease AsP Is a Short Secretory Isoform of the Transmembrane Airway Trypsin-Like Protease;Endocrinology;2004-04-01
4. N-Terminal Proopiomelanocortin Acts as a Mitogen in Adrenocortical Tumor Cells and Decreases Adrenal Steroidogenesis;The Journal of Clinical Endocrinology & Metabolism;2003-05
5. Acute in vivo Effects of Acth by Exo Utero Microinjection on Differentiation, Steroidogenesis and Proliferation of Fetal Mouse Adrenocytes;Endocrine Research;1999-02
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