BCR-ABL1 kinase domain mutation analysis by next generation sequencing detected additional mutations in chronic myeloid leukemia patients with suboptimal response to imatinib
Author:
Affiliation:
1. Department of Hematology, Christian Medical College, Vellore, India
2. MedGenome Labs Ltd, Bengaluru, India
Funder
Centre of Excellence grant from Department of Biotechnology India
Indian Council of Medical Research Centre for Advanced Research
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
https://www.tandfonline.com/doi/pdf/10.1080/10428194.2021.1872074
Reference14 articles.
1. Poor response to second-line kinase inhibitors in chronic myeloid leukemia patients with multiple low-level mutations, irrespective of their resistance profile
2. BCR-ABL1 kinase domain mutations may persist at very low levels for many years and lead to subsequent TKI resistance
3. Next-generation deep sequencing improves detection of BCR-ABL1 kinase domain mutations emerging under tyrosine kinase inhibitor treatment of chronic myeloid leukemia patients in chronic phase
4. Plasma imatinib levels and ABCB1 polymorphism influences early molecular response and failure-free survival in newly diagnosed chronic phase CML patients
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1. Imatinib-resistant Chronic Myeloid Leukaemia Patients' BCR-ABL1 Kinase Domain Mutations in the Eastern Indian Population;2024-04-02
2. Analysis of Two Mutations in the BCR-ABL Fusion Gene Relevant for Monitoring Chronic Myeloid Leukemia Patients;Biointerface Research in Applied Chemistry;2022-07-12
3. Deep sequencing reveals the spectrum of BCR-ABL1 mutations upon front-line therapy resistance in chronic myeloid leukemia: An Eastern-Indian cohort study;Cancer Treatment and Research Communications;2022
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