Monomeric α-synuclein (αS) inhibits amyloidogenesis of human prion protein (hPrP) by forming a stable αS-hPrP hetero-dimer.-Reference-Cited by-同舟云学术

Monomeric α-synuclein (αS) inhibits amyloidogenesis of human prion protein (hPrP) by forming a stable αS-hPrP hetero-dimer.

Published:2021-01-01 Issue:1 Volume:15 Page:37-43
ISSN:1933-6896
Container-title:Prion
language:en
Short-container-title:Prion

Author:

Yamashita Satoshi¹,O. Kamatari Yuji¹²,Honda Ryo¹^ORCID,Niwa Ayumi³,Tomiata Hiroyuki³,Hara Akira³,Kuwata Kazuo¹⁴

Affiliation:

1. United Graduate School of Drug Discovery and Medical Information Sciences, Gifu University, Tokai National Higher Education and Research System, Gifu, Japan

2. Institute for Glyco-core Research, Tokai National Higher Education and Research System, Gifu, Japan

3. Department of Tumor Pathology, Gifu University Graduate School of Medicine, Tokai National Higher Education System, Gifu, Japan

4. Department of Gene and Development, Gifu University School of Medicine, Tokai National Higher Education and Research System, Gifu, Japan

Publisher

Informa UK Limited

Subject

Infectious Diseases,Cell Biology,Cellular and Molecular Neuroscience,Biochemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/19336896.2021.1910176

Reference35 articles.

1. Protein Misfolding, Functional Amyloid, and Human Disease

2. Synthetic Mammalian Prions

3. NMR studies in aqueous solution fail to identify significant conformational differences between the monomeric forms of two Alzheimer peptides with widely different plaque-competence, Aβ(1-40)oxand Aβ(1-42)ox

4. Molecular Crowding Accelerates Fibrillization of α-Synuclein: Could an Increase in the Cytoplasmic Protein Concentration Induce Parkinson's Disease?

5. Molecular Biology of Prion Diseases

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1. Roles of Apigenin and Nepetin in the Assembly Behavior and Cytotoxicity of Prion Neuropeptide PrP106-126;ACS Chemical Neuroscience;2023-12-22

2. Spatiotemporal modulations in heterotypic condensates of prion and α-synuclein control phase transitions and amyloid conversion;Nature Communications;2022-03-03